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VIP The intestinal peptide
VIP is reported as an anti-inflammatory and immune-modulatory peptide that has considered being a potential candidate for treatment of inflammatory and autoimmune diseases through down-regulation of inflammatory cytokines and mediators. VIP helps modulate effective communication in the immune system with the rest of the body. This makes VIP a key player in your immune and gut health. There is mounting evidence that increasing VIP levels can combat and even reverse some of the effects of autoimmune and degenerative disorders.
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BPC-157 stomach, gastric and injury repair peptide
Research has shown BPC-157 to have considerable biological healing properties. Experimentally it has been demonstrated to accelerate the healing of many different wounds, including tendons, muscles, nervous system and superior healing of damaged ligaments. This peptide can be very useful to athletes or just the average person who has daily aches, pains or injury. Overuse of our body can cause muscle, tendon and ligament issues that affect our everyday life. The addition of BPC-157 can help heal those everyday aches and pains
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Thymosin beta-4 injury repair peptide
TB-500 is a synthetic fraction of the 43 amino acid protein thymosin beta-4. The main purpose of this peptide is to promote healing. The healing effects of TB-500 have been observed in tendons, ligaments, muscles, skin, heart, and eyes. It aids in the quick healing of wounds by promoting new blood vessel and muscle fiber growth. It upregulates cell building proteins like actin. It is fast acting and goes to the site of the injury once injected and starts to work.
Learn moreThymosin Alpha 1 immunoprotectant peptide
Thymosin alpha 1 (T α 1) is a peptide, or small protein, produced naturally by the thymus gland. The thymus is where immune cells known as T cells mature and are released when prompted to do so by the T α 1 peptide. T cell production and action within the body is vital to adaptive immunity. This is the mode by which immune cells are able to recognize and kill foreign invaders. Specifically, T α 1 has been shown to enhance the function of certain immune cells called T and dendritic cells. These white blood cells play pivotal roles in the body’s defense process to anyone with a depressed immune system or suffering from an infection. Thymosin alpha 1 has three hallmark actions. It can improve immune modulation, it can be immune stimulating, or it can be immune dampening. It takes advantage of our innate and acquired immune system and does what is necessary depending on what is needed. Benefits of Thymosin alpha 1: Enhances the function of certain immune cells called T and dendritic cells Effective for acute and chronic infections Help eradicate the unhealthy or senescent cells and stops the infection or cancer growth Exhibits antibacterial, antiviral and antifungal properties Suppresses tumor growth Increases vaccine effectiveness Protects against oxidative damage T cells, for example, come in two forms: killer and helper T cells. Killer T cells are responsible for hunting down and destroying our body’s own cells that are cancerous or infected with bacteria or viruses. Helper cells work with the other cells of the immune system to orchestrate and carry out appropriate immune responses. It is approved in more than 37 countries for the treatment of hepatitis B, hepatitis C, and as an adjunct to chemotherapy and various vaccines. T α 1 has been found to have a profound effect on the immune system and is the active ingredient in the immune modulating drug, Zadaxin®. Zadaxin® is used to treat hepatitis B and C and has been studied extensively for its ability to support an immune system that has been suppressed by chemotherapy in cancer patients. Additional possible indications are malignant melanoma, hepatocellular carcinoma, drug-resistant tuberculosis, chronic fatigue and Di George's syndrome as well as any chronic cancer or viral disease. In addition to its use in the treatment of Hepatitis, AIDS, and cancer, T α 1 has shown great promise in the treatment of Lyme disease. This is unsurprising when one considers the large role the immune system plays in combating the disease. T α 1 assists the immune system in the location and eradication of the Lyme bacteria and infected cells, while helping to prevent oxidative damage, thereby decreasing inflammation and enabling a better quality of life throughout treatment. Senomodulator Thymosin alpha 1 is also a natural senolytic ( induce death of aging/senescent cells) that our thymus makes. It decreases with age because of thymic atrophy and this problem affects the production of thymosin alpha 1. Thymosin alpha 1 can help bring back natural killer cell function and influence production of interleukin 1 beta, tumor necrosis factor, interleukin 6 and nuclear factor kappa B. It is also a senolytic because it helps senescent cells become more recognizable by macrophages and other cells in the immune system. Thymosin alpha 1 can activate and modulate the immune system to become a natural senolytic because in reality it's natural to the system. This is how Thymosin Alpha 1 acts as a senomodulator. Thymosin Research: Studies have shown that individuals fighting infection have a lower amount of circulating T α 1 and suppressed helper T cell numbers compared to healthy individuals. This is problematic, as the optimal immune function is vital to recovery from infection. Supplementation with T α 1 has the potential for great therapeutic benefit for patients looking to strengthen their immune system and those suffering from an infection or autoimmune disease. Immune Modulation with Thymosin Alpha 1 Treatment.pdf Pica, F., Chimenti, M. S., Gaziano, R., Buè, C., Casalinuovo, I. A., Triggianese, P., … Garaci, E. (2016). Serum thymosin α 1 levels in patients with chronic inflammatory autoimmune diseases. Clinical & Experimental Immunology, 186(1), 39–45 Bozza, S., Gaziano, R., Bonifzi, P., et al. (2007). Thymosin alpha 1 activates the TLR9/MyD88/IRF7-dependent murine cytomegalovirus sensing for induction of anti-viral responses in vivo. International Immunology, 19, 1261–1270. Chan, H. L., Tang, J. L., & Sung, J. Y. (2001). Thymosin a1 for the treatment of chronic hepatitis B virus (HBV) infection: A meta-analysis. In Paper presented at the digestive disease week 2001; May 20–23, Atlanta, GA. Chen, J. (2007). Effects of thymosin α1 on cell immunity function in patients with septic shock. Zhongguo Wei Zhong Bing Ji Jiu Yi Xue, 19, 153–155 Knutsen, A. P., Freeman, J. J., Mueller, K. R., Roodman, S. T., & Bouhasin, J. D. (1999).Thymosin-alpha1 stimulates maturation of CD34+ stem cells into CD3+4+ cells in an in vitro thymic epithelia organ coculture model. International Journal of Immunopharmacology, 21(1), 15–26. Li, C., Wang, C.-H., Meng, Q.-H., Ye, S.-L., Wang, X.-J., & Jiang, C. (2007). Effect of the thymosin alpha 1 on immune function in aged chronic obstructive pulmonary disease during acute period. Chinese Journal of Hospital Pharmacy, 27(5), 637–639. Romani, L., Bistoni, F., Gaziano, R., et al. (2004). Thymosin alpha 1 activates dendritic cells for antifungal Th1 resistance through toll-like receptor signaling. Blood, 103(11), 4232–4239. Serafino, A., Pica, F., Andreola, F., Gaziano, R., Moroni, N., Moroni, G., et al. (2014). Thymosin α1 activates complement receptor-mediated phagocytosis in human monocytederived macrophages. Journal of Innate Immunity, 6(1), 72–88. Serafino, A., Pierimarchi, P., Pica, F., et al. (2012). Thymosin alpha 1 as a stimulatory agent of innate cell-mediated immune response. Annals of the New York Academy of Sciences, 1269–1270, 43–50. Serrate, S., Schulof, R., Leondaridis, L., Goldstein, A. L., & Sztein, M. B. (1987). Modulation of human natural killer cell cytotoxic activity, lymphokine production, and interleukin 2 receptor expression by thymic hormones. Journal of Immunology, 139, 2338–2343.
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MOTS-C Mitochondrial fed weight loss
Research also shows that skeletal muscle is the major target tissue of MOTS‐c. The skeletal muscle enhances insulin sensitivity and increases glucose uptake in myocytes (muscle cells) by activating the AMPK pathway and at the same time without increasing insulin. It is fair to call MOTS‐c an exercise‐mimetic, meaning it imitates exercise on the body. Exercise also increases muscle glucose uptake without stimulating insulin.
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5 Amino mq 1 for NNMT weight loss
Benefits of 5-amino 1MQ Can reverse diet-induced obesity Can treat related metabolic conditions Can increase cellular energy regulators Can prevent lipogenesis (fat accumulation) Can increase NAD+and SAM concentrations in fat cells Can regulate energy expenditure in fat cells
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AOD9604 the anti obesity drug
AOD-9604 activates degradation and fat burning (oxidation) by a method that doesn't use the hGH receptor; it has its own mechanism of action independent of hGH. Laboratory studies on rodents, pigs, dogs & humans have identified the AOD-9604 mechanism which triggers fat release from the obese fat cells predominantly and works to reduce the accumulation of new fat in fat cells as well as increase fat burning. What is extraordinary about the AOD-9604 is its ability to reduce abdominal fat stores.
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Skincare peptide Ghk-cu
GHK-Cu can promote activation of wound recovery, appeal of immune cells, antioxidant, and anti-inflammatory consequences, stimulation of collagen and glycosaminoglycan synthesis in pores and skin fibroblasts and merchandising of blood vessels increase. GHK is a safe, extensively studied compound that has a wealth of positive and health-promoting effects in many tissues and systems. It has been widely used in anti-aging and cosmetic products in humans for decades without any adverse effects and can be easily incorporated in creams, liposomes, foams or subcutaneous injections.
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Melanotan the tan in a vial
Clinical trials have shown that use of Melanotan II may hold the potential to promote melanogenesis, with minimal side effects. The primary role of melanogenesis is to protect the hypodermis, which is the layer under the skin from the UV-B light that causes damage. It works by absorbing all of the UV-B light, which blocks its passage into the skin layer.
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